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In addition to the traditionally recognized mechanisms of peripheral nerve damage and central sensitization, excessive iron accumulation, oxidative stress, neuronal inflammation, and lipid peroxidation damage are distinctive features of NP in pathophysiology

Based on the findings that wild-type (WT) p53 represses SLC7A11 expression and p53 deficiency promotes SLC7A11 expression in cancer cells (Jiang et al., 2015), one would expect that p53 mutants with oncogenic functions, similar to p53 deficiency, should promote SLC7A11 expression

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