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Studies that deleted either MECP2 _e1 or e2 throughout the body found that only deletion of MECP2 _e1 led to RTT-like phenotypes and a shortened lifespan, while deletion of e2 was required for placental development (Itoh et al

Previously reported representative rare metabolic disorders included galactosialidosis [9], biotinidase deficiency [10], Wolmans disease [11], lysinuric protein intolerance [12], NiemannPick disease [13], Gaucher disease [14], propionic acidemia [3], Pearson syndrome [15], and multiple sulphatase deficiency [16]

S, puedes, pero con cuidado