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doi: 10.1016/j.autrev.2009.02.020

In vitro, BPC-157 promotes fibroblast migration, angiogenesis, and nitric oxide (NO) pathway modulation, supporting endothelial cell survival and wound repair

Rather than targeting a single mechanism, it simultaneously promotes blood vessel formation (angiogenesis), enhances growth factor signaling, increases nitric oxide for better tissue oxygenation, and suppresses inflammatory markers

Recent studies show that exposure to GLP1-RA in the first trimester is not associated with a risk of major birth defects when compared with diabetes or obesity