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Therefore, cancer cells may be more susceptible to ALT activation due to the binding of epitalon to the already reduced levels of histone H1, whereas normal cells may be more resilient as they naturally express higher levels of H1

Notably, while some subunits of spliceosome complexes normally recruited during its activation (e.g., NTR, NTC, IBC) were still incorporated (e.g., SNW1, RBM22, PRPF19, AQR), the others were decreased (e.g., BCAS2, CDC5L, CWC22)

Clinical Research and Evidence Learn more about the research supporting the effectiveness of AOD-9604: How to Begin AOD-9604 Therapy 1

DOI: 10.1016/j.jchromb.201506.007 88