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found that I/R was able to lead to liver damage, LPO as well as upregulation of the ferroptosis marker Ptgs2 in a murine model, and all of these effects were significantly alleviated by ferroptosis-specific inhibitor ferrostatin-1 and iron chelator deferoxamine [18]

[DOI] [PubMed] [Google Scholar] 362.Jin Z, Dridi N, Palui G, Palomo V, Jokerst JV, Dawson PE, Amy Sang Q-X and Mattoussi H, J

A.GentryC.LightE.VastaniN.VallortigaraJ.BierhausA.et al

The resulting catastrophic energy deficit is a primary physiological driver of post-exertional malaise (PEM) or 'crashes,' where even minor, everyday activities trigger a disproportionate and prolonged exacerbation of profound fatigue, muscle weakness, and cognitive impairment