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Although in both disorders the immune response is different, genetically susceptible individuals have an inappropriate mucosal immune response against their gut microbiota, mediated by an excessive activation of effector T cell subsets and/or a deficiency of Treg cells (65) (Figure 1A)

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Furthermore, because oxygen radicals can release iron from iron-sulfur clusters, factors that promote regeneration of iron-sulfur clusters can also play a role in recovering from oxidative damage

Thus, POAG progression seems to be associated with the accumulation of zinc-amino acid complexes, especially in the early stages of the disease