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Excessive alcohol consumption results in a range of alcoholic liver disease (ALD), from simple alcoholic fatty liver to advanced alcoholic hepatitis, cirrhosis, and eventually hepatocellular carcinoma [2]

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This increased conversion promotes the flux of the TCA cycle to meet the synthetic and metabolic demands of tumor cells and simultaneously regulates serine hydroxymethyltransferase 1 (SHMT1) activity to promote the serineglycine metabolic axis and one-carbon unit supply

For injury recovery (BPC-157, TB-500), animal studies show promise, but human clinical evidence is essentially nonexistent